Researchpublished in the Journal of Neurosciencehas key a potential room to stop the cognitive diminution relate with Alzheimer ’s , and has shown substantial hope with mouse models . Here ’s how it works .
The discourse practice antibodies to target one specific ( and competently named ) protein called Dickkopf-1 ( Dkk1 ) . See , one of the characteristics of Alzheimer ’s disease abnormally in high spirits deposits in the mental capacity of the protein Amyloid - ß — which is connect to damage to the synapsis . Patients with Alzheimer ’s have increased stage of Dkk1 in their brains . The investigator believe that Amyloid - ß causes the yield of Dkk1 , which is what dismantles the synapses in the genus Hippocampus , damage your computer storage .
By looking at nous slices of mice , the investigator were able to get across the State Department of the mice ’s synapses , both in creature exposed to Amyloid - ß , and those also treat with specific Dkk1 antibody . The intervention protected the brains , and leave in no synaptic disintegration .
Now , it ’s a tenacious road between mice and humans , and this wo n’t function to aid anyone who has already suffered from the disease . But it does potentially reveal some of Alzheimer ’s mechanism , and potentially , when combined with other warnings , may be able to prevent cognitive decay before it starts .
Alzheimer ’s diseaseMedicineNeuroscienceScience
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